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Genetic inactivation of calpain-1 attenuates pain sensitivity in a humanized mouse model of sickle cell disease

Graduate Program in Pharmacology and Experimental Therapeutics, Sackler School of Graduate Biomedical Sciences, Tufts University School of Medicine, Boston, MA
Vascular Biology Center, Division of Hematology, Oncology and Transplantation, Department of Medicine, University of Minnesota, Minneapolis, MN
Children’s Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX
Department of Laboratory Medicine, Boston Children’s Hospital and Department of Pathology, Harvard Medical School, Boston, MA
Vascular Biology Center, Division of Hematology, Oncology and Transplantation, Department of Medicine, University of Minnesota, Minneapolis, MN
Graduate Program in Pharmacology and Experimental Therapeutics, Sackler School of Graduate Biomedical Sciences, Tufts University School of Medicine, Boston, MA;Department of Developmental, Molecular and Chemical Biology, Tufts University School of Medicine, Boston, MA, USA
Vol. 101 No. 10 (2016): October, 2016 https://doi.org/10.3324/haematol.2016.148106